Saeki
A, Tsuchiya K, Suda T, Into T, Hasebe A, Suzuki T, and Shibata KI. Gasdermin
D-independent release of interleukin-1 by living macrophages in response to
mycoplasmal lipoproteins and lipopeptides. Immunology. 2020, 161:114-122
Mahib
MR, Hosojima S, Kushiyama H, Kinoshita T, Shiroishi T, Suda T, and Tsuchiya K.
Caspase-7 mediates caspase-1-induced apoptosis independently of Bid. Microbiol. Immunol. 2020,
64(2):143-152.
Dodo
K, Kuboki E, Shimizu T, Imamura R, Magarisawa M, Takahashi M, Tokuhiro T,
Yotsumoto S, Asano K, Nakao S, Terayama N, Suda T, Tanaka M, and Sodeoka M.
Development of a Water-Soluble Indolylmaleimide Derivative IM-93 Showing Dual
Inhibition of Ferroptosis and NETosis. ACS Med Chem Lett.
2019;10(9):1272]1278.
Watanabe
Y, Nagai Y, Honda H, Okamoto N, Yanagibashi T, Ogasawara M, Yamamoto S, Imamura
R, Takasaki I, Hara H, Sasahara M, Arita M, Hida S, Taniguchi S, Suda T, and
Takatsu K. Bi-directional crosstalk between neutrophils and adipocytes promotes
adipose tissue inflammation. FASEB
J. 2019, 33:11821-11835
Fang
R, Uchiyama R, Sakai S, Hara H, Tsutsui H, Suda T, Mitsuyama M, Kawamura I, and
Tsuchiya K. ASC and NLRP3 maintain innate immune homeostasis in the airway
through an inflammasome-independent mechanism. Mucosal
Immunol. 2019, 12:1092-1103.
Tsuchiya
K, Nakajima S, Hosojima S, Nguyen DT, Hattori T, Le TM, Hori O, Mahib MR,
Yamaguchi Y, Miura M, Kinoshita T, Kushiyama H, Sakurai M, Shiroishi T, and
Suda T. Caspase-1 initiates apoptosis in the absence of gasdermin D. Nat
Commun, 2019, 10:2091. doi: 10.1038/s41467-019-09753-2.
Nakajima
S, Imamura R, Yoshino M, Sakurai M, Tsuchiya K, Sugihara K, Asano M, and Suda
T. Characterization 1 of Innate and Adaptive Immune Responses in
PYNOD-deficient mice. ImmunoHorizons,
2018, 2:129-141. doi: 10.4049/immunohorizons.1700074
Kamikawa
Y, Sakai N, Miyake T, Sagara A, Shinozaki Y, Kitajima S, Toyama T, Hara A,
Iwata Y, Shimizu M, Furuichi K, Imamura R, Suda T, Kaneko S, Wada T.:
Involvement of p38MAPK in Impaired Neutrophil Bactericidal Activity of
Hemodialysis Patients. Ther
Apher Dial. 2018, 22(4):345–354
Zhang
P, Tsuchiya K, Kinoshita T, Kushiyama H, Suidasari S, Hatakeyama M, Imura H,
Kato N, Suda T.: Vitamin B6 prevents IL-1Ŕ production
by inhibiting NLRP3 inflammasome activation. J
Biol Chem. 2016, 291:24517-24527
Kinoshita,
T., Imamura, R., Kushiyama, H., and Suda, T.: NLRP3 mediates NF-ČB activation and cytokine induction in microbially induced and
sterile inflammation. PLoS
One. 2015, 10(3):e0119179
Wang,
Q., Imamura, R., Motani, K., Kushiyama, H., Nagata, S., and Suda, T. Pyroptotic cells externalize eat-me and
release find-me signals and are efficiently engulfed by macrophages. Int. Immunol. 2013,
25(6):363-372.
Harashima, N., Inao, T., Imamura, R., Okano, S., Suda, T.,
and Harada, M.: Roles of
the PI3K/Akt pathway and autophagy in TLR3 signaling-induced apoptosis and
growth arrest of human prostate cancer cells. Cancer
Immunol. Immunother. 2012, 61(5):667-676
Motani K, Kushiyama H, Imamura R,
Kinoshita T, Nishiuchi T, and Suda T.FCaspase-1 induces apoptosis-associated speck-like protein containing
a caspase-recruitment domain (ASC)-mediated necrosis independently of its
catalytic activity. J Biol Chem. 2011
Motani, K., Kawase, K., Imamura, R., Kinoshita, T., Kushiyama, H., and Suda, T.: Activation of ASC
induces apoptosis or necrosis, depending on the cell type, and causes tumor eradication. Cancer Sci.,
2010, 101(8):1822-1827.
Imamura, R., Wang, Y., Kinoshita, T.,
Suzuki, M., Noda, T.,
Sagara, J., Taniguchi, S., Okamoto, H., and Suda, T.: Anti-inflammatory activity of PYNOD and its mechanism
in humans and mice. J. Immunol., 2010, 184(10):5874-5884.
Hasegawa,
M., Imamura, R., Motani, K., Nishiuchi, T., Matsumoto, N., Kinoshita, T., and
Suda, T.: Mechanism and repertoire of ASC-mediated gene expression. J. Immunol., 2009, 182(12):7655-7662.
Ohno,
S.,@Kinoshita, T., Ohno, Y., Minamoto, T., Suzuki N.,
Inoue M., and Suda, T.: Expression of NLRP7 (PYPAF3, NALP7) protein in
endometrial cancer tissues. Anticancer Res., 2008, 28(4C):2493-2497.
Matsumoto,
N., Imamura, R., Suda, T.: Caspase-8- and JNK-dependent AP-1 activation is
required for Fas ligand-induced IL-8 production. FEBS
J. 2007, 274:2376-2384.
Umemura, M., Yahagi, A., Hamada, S., Begum,
M. D., Watanabe, H., Kawakami, K., Suda, T., Sudo, K., Nakae, S., Iwakura, Y.,
and Matsuzaki, G.: IL-17-mediated regulation of innate and acquired immune
response against pulmonary Mycobacterium bovis BCG infection, J.
Immunol., 2007, 2007, 178:3786-3796.
Hasegawa, M., Kawase, K., Inohara, N., Imamura, R., Yeh,
W-C.,Kinoshita, T., and Suda, T.: Mechanism of
ASC-mediatedapoptosis: Bid-dependent apoptosis in type II cells, Oncogene,
2007,
26:1748-1756.
Fujisawa, A., Kambe, N., Saito, M.,
Nishikomori, R., Tanizaki, H., Kanazawa, N., Adachi, S., Heike, T., Sagara, J.,
Suda, T., Nakahata, T., Miyachi, Y.: Disease-associated mutations in CIAS1
induce cathepsin B-dependent rapid cell death of human THP-1 monocytic cells, Blood,
2007,
109:2903-2911.
Kinoshita, T., Kondoh, C., Hasegawa, M.,
Imamura, R., and Suda, T.: Fas-associated Factor 1 is a negative regulator of
PYRIN-containing Apaf-1-like protein 1, Int.
Immunol., 2006, 18:1701-1706.
El Kasmi, K.C., Holst J, Coffre, M.,
Mielke, L., de Pauw, A., Lhocine, N., Smith, A.M., Rutschman, R., Kaushal, D.,
Shen, Y., Suda, T., Donnelly, R.P., Myers, M.G. Jr., Alexander, W., Vignali,
D.A., Watowich, S.S., Ernst, M., Hilton, D.J., Murray, P.J.: General nature of
the STAT3-activated anti-inflammatory response. J.
Immunol., 2006, 177:7880-7888.
Kidoya, H., Umemura, U., Kawabe, T., Matsuzaki, G., Yahagi, A., Imamura,R., and Suda, T.: Fas ligand induces cell-autonomous IL-23 production indendritic cells, a mechanism for Fas ligand-induced IL-17 production. J.Immunol., 2005, 175:8024-31.
Kinoshita, T., Wang, Y., Hasegawa, M., Imamura, R., and Suda, T.: PYPAF3, a PYRIN-containing APAF-1-like protein, is a feedback regulator ofcaspase-1-dependent interleukin-1˛ secretion. J.Biol. Chem., 2005, 280:21720-5
Hasegawa M, Imamura R, Kinoshita T, Matsumoto N, Masumoto J, Inohara N,and Suda T.: ASC-mediated NF-şB activation leading to interleukin-8production requires caspase-8 and is inhibited by CLARP. J.Biol. Chem., 2005, 280: 15122-30.
Imamura, R., Konaka, K., Matsumoto, N., Hasegawa, M.,
Umemura, M., Kawabe, T., Shudo, K., Kidoya, H.,
Wang, Y., Hasegawa, M., Imamura, R., Kinoshita, T., Kondo, C., Konaka,K., Suda, T.: PYNOD, a novel Apaf-1/CED4-like protein is an inhibitor ofASC and caspase-1. Int.Immunol., 2004, 16:777-86.
Nakamoto Y, Suda T, Momoi T, and Kaneko S.: Differentprocarcinogenic potentials of lymphocyte subsets in a transgenic mouse model ofchronic hepatitis B. Cancer Res., 2004, 64:3326-3333
Tachiiri, A., Imamura, R., Wang, Y., Fukui, M., Umemura, M., and Suda, T.: Genomic structure and inducible expression of the IL-22 receptor alpha chain in mice. Gene. Immun., 2003, 4:153-159
Nakamoto, Y. Kaneko, S., Fan, H., Momoi, T., Tsutsui, H., Nakanishi, K.,Kobayashi, K., and Suda, T.: Prevention of Hepatocellular CarcinomaDevelopment Associated with Chronic Hepatitis by Anti-Fas Ligand AntibodyTherapy. J. Exp. Med., 2002, 196:1105-1111
Fukuyama H, Adachi M, Suematsu S, Miwa K, Suda T, Yoshida N, Nagata S.: Requirement of Fas expression in B cells for tolerance induction. Eur. J. Immunol., 2002, 32:223-30.
Shudo, K., Kinoshita, K., Imamura, R., Fan, H., Hasumoto, K., Tanaka, M.,Nagata, S., and Suda, T.: The membrane-bound but not the soluble form ofhuman Fas ligand is responsible for its inflammatory activity. Eur.J. Immunol., 2001, 31:2504-2511
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